Sunday, 18 February 2018

ORAL SUB MUCOUS FIBROSIS : Physiotherapy Treatment

A Patient with Oral submucosal Fibrosis


Oral submucous fibrosis is characterized as the unending, tricky ailment influencing the oral pit and here and there pharynx, albeit at times went before or potentially connected with vesicle arrangement and is constantly connected with juxtaepithelial fiery response took after by fibro versatile changes in the lamina propria with epithelial decay prompting firmness of oral pit prompting trismus and powerlessness to eat.

Oral submucous fibrosis is an interminable crippling and an all around perceived possibly threatening condition related with areca nut biting, an element of betel quid and is pervasive in South Asian populace. Pathogenesis isn't yet settled however is accepted to be because of multifactorial causes; consequently the treatment of oral submucous fibrosis proposes a noteworthy test for oral doctors.


The pathogenesis of the illness isn't entrenched, however the reason for OSF is accepted to be multifactorial.

Various variables may trigger the infection procedure by causing a juxtaepithelial fiery response in the oral mucosa. Components incorporate are areca nut biting, ingestion of chilies, hereditary and immunologic procedures, wholesome inadequacies and different variables.

Areca Nut (Betel Nut) Chewing:

The areca nut segment of betel quid assumes a noteworthy part in the pathogenesis of OSF 15. Betel nut is much of the time utilized as a psychotropic and antihelminthic operator and utilized as an after feast digestant which is taken to ease stomach inconvenience.

Smoking and liquor utilization alone, propensities basic to areca nut chewers, have been found to have no impact in the advancement of OSF. The most grounded confirm in regards to the etiology of OSF is with the propensity for areca nut biting.

Areca nut shape might be accessible in thefollowing structure:

Supari + Tobacco

Supari + Pan+ Tobacco

Supari + Pan + Pan masala

Skillet Parag/Pan masala

Supari + Pan + Lime

Supari-Roasted/Raw Areca nut

Part of areca nut in pathogenesis of OSF:

Arecoline, a dynamic alkaloid found in betel nuts. Animates fibroblasts to build creation of collagen by 150%.

To lift the mRNA and protein articulation of cystatin C, a nonglycosylated fundamental protein reliably up-directed the assortment of fibrotic illnesses, in a measurement subordinate way in people with OSF.

Areca nuts have likewise been appeared to have a high copper substance, and biting areca nuts for 5-30 minutes altogether increments solvent copper levels in oral liquids. This expanded level of solvent copper underpins the speculation as a starting element in people with OSF.

Healthful Deficiencies:

Press insufficiency frailty, vitamin B complex inadequacy and lack of healthy sustenance are advancing variables that unsettle the repair of the excited oral mucosa, prompting damaged recuperating and resultant scarring.

The resultant atrophic oral mucosa is more defenseless to the impacts of chilies and betel nuts. Mucosal changes like those in vitamin B and iron insufficiency are found in oral sub mucosal fibrosis.


The part of chillies ingestion in the pathogenesis of OSF is disputable.

A touchiness response to chilies is accepted to add to OSF.

Hereditary and Immunologic Processes:

A hereditary segment is thought to be engaged with OSF Patients with expanded recurrence of HLA-A10, HLA-B7, and HLA-DR3.

Phases OF OSF:

Stage 1:

Stomatitis incorporates erythematous mucosa, vesicles, mucosal ulcers, melanotic mucosal pigmentation, and mucosal petechia.

Stage 2:

Fibrosis happens in cracked vesicles and ulcers when they mend, which is the sign of this stage.

Early sores show whitening of the oral mucosa.

More established sores incorporate vertical and round unmistakable sinewy groups in the buccal mucosa and around the mouth opening or lips, bringing about a mottled, marble like appearance of the mucosa in view of the vertical, thick, stringy groups running in a whitening mucosa. Particular discoveries incorporate the accompanying:

Decrease of the mouth opening (trismus).

Solid and little tongue.

Whitened and rough floor of the mouth.

Fibrotic and depigmented gingiva.

Rubbery delicate sense of taste with diminished portability.

Whitened and atrophic tonsils.

Contracted budlike uvula.

Sinking of the cheeks, not comparable with age or nutritious status.

Stage 3:

Screech of OSF are as per the following:

Leukoplakia is precancerous and is found in over 25% of people with OSF.

Discourse and hearing deficiencies may happen in view of inclusion of the tongue and the eustachian tubes.



Intermittent ulceration.

Torment in the ear or deafness.

Nasal pitch of voice.

Confinement of the development of the delicate sense of taste.

Diminishing and hardening of the lips.

Pigmentation of the oral mucosa.

Dryness of the mouth and consuming sensation.

Diminished mouth opening and tongue bulge.


Mouth Opening Device in SMF

Immunological ailments.

Outrageous climatic conditions.

Delayed insufficiency to iron and vitamins in the eating regimen.


Oral appearances of scleroderma

Oral appearances of Plummer Vinson disorder (Iron lack Anemia).


Finish Hemogram

Toludine blue test

Biopsy :- Incisional biopsy

Immunofluorescent test:

a) Direct b) Indirect

Administration AND PREVENTION:

The treatment of patients with OSF relies upon the level of clinical contribution. On the off chance that the malady is identified at a beginning period, suspension of the propensity is adequate. Most patients with OSMF give moderateto-serious arranging. Direct to-serious arranging of OSF is irreversible. 

Medicinal treatment is symptomatic and gone for enhancing mouth developments.

Not to devour areca nut and other incessant aggravation, for example, hot and zesty sustenance including chiles.

Guidance green verdant vegetables.

Organization of Vit. A, B complex and high protein consume less calories.

Organization of Antoxid OD for 6 – two months.

Organization of Lycored OD for 6 two months.

Keeping up legitimate oral cleanliness.

Supplementing the eating regimen with nourishments rich in vitamins A, B complex, and C and iron.

Swearing off hot liquids like tea, espresso.

Swearing off liquor.

Utilizing a dental specialist to round off sharp teeth and concentrate third molars.


Surgical treatment is shown in patients with extreme conditions. These incorporate:-

Basic extraction of the stringy groups: Excision can bring about contracture of the tissue and intensification of the condition.

Split-thickness skin joining following respective temporalis myotomy or coronoidectomy: Trismus related with OSF might be because of changes in the temporalis ligament auxiliary to OSF; subsequently, skin unions may assuage.

Nasolabial folds and lingual pedicle folds: Surgery performed just in patients with OSF in whom the tongue isn't included.


Mouth Opening Exercise In SMF


Muscle extending practices for the mouth might be useful to anticipate advance restrictions of mouth opening strong mouth opening has been attempted with mouth choke and non-cyclic surgical screw.

Diathermy: Microwave diathermy appear to be better than short wave, in light of the fact that specific warming of juxtaepitheliel connective tissue is conceivable it acts by physio fibrinolysis of groups.

Ultrasound: Ultra sound selectivity bring the temperature up in some all around aggregated territories. Ultrasound turns out to be an effective profound warming methodology.

Monday, 22 January 2018

OSTEOPOROSIS : Exercise And Diet


                      PARALYSIS,SPACE TRAVEL ETC.
                     CHRONIC ALCOHOLISM

Wednesday, 10 January 2018

Bicipital Tendinitis And Physiotherapy Treatment : Overview

Biceps tendinitis is inflammation of the tendon around the long head of the biceps muscle.
Bicipital Tendinitis


Biceps tendinosis is caused by degeneration of the tendon from athletics requiring overhead motion or from the normal aging process. 

Anatomy Of Bicipital Tendinitis


Inflam-mation of the biceps tendon in the bicipital groove, which is known as primary biceps tendinitis, occurs in 5 percent of patients with biceps tendinitis.
Biceps tendinitis and tendinosis are commonly accompanied by rotator cuff tears or SLAP (superior labrum anterior to posterior) lesions. Patients with biceps tendinitis or tendinosis usually complain of a deep, throbbing ache in the anterior shoulder. Repetitive overhead motion of the arm initiates or exacerbates the symptoms. The most common isolated clinical finding in biceps tendinitis is bicipital groove point tenderness with the arm in 10 degrees of internal rotation. Local anesthetic injections into the biceps tendon sheath may be therapeutic and diagnostic. Ultrasonography is preferred for visualizing the overall tendon, whereas magnetic resonance imaging or computed tomography arthrography is preferred for visualizing.the intraarticular tendon and related pathology. Conservative management of biceps tendinitis consists of rest, ice, oral analgesics, physical therapy, or corticosteroid injections into the biceps tendon sheath. Surgery should be considered if conservative measures fail after three months, or if there is severe damage to the biceps tendon.

Anatomy and Physiology:

The long head of the biceps tendon rises from the supraglenoid tubercle and the superior glenoid labrum.
The proximal portion of the long head of the biceps tendon is extrasynovial but intra-articular.
5 The tendon travels obliquely inside the shoulder joint, across the humeral head anteriorly, and exits the joint within the bicipital groove of the humeral head beneath the transverse humeral ligament.
 The bicipital groove is defined by the greater tuberosity (lateral) and the lesser tuberosity (medial). The biceps tendon is contained in the rotator interval, a triangular area between the subscapularis and supraspinatus tendons at the shoulder (Figure 1). The rotator interval is responsible for keeping the biceps tendon in its correct location.6–8 Because the rotator interval is usually indistinguishable from the rotator cuff and capsule, lesions of the biceps tendon are usually accompanied by lesions of the rotator cuff

SLAP lesions are often present in patients with biceps tendinitis and tendinosis. The anterosuperior labrum and superior labrum are more likely to tear than the inferior portion of the labrum because they are not attached as tightly to the glenoid.9–13 Additionally, certain conditions that affect the glenohumeral joint may also involve the biceps tendon because it is intra-articular. These may include rheumatologic (e.g., rheumatoid arthritis, lupus), infectious, or other types of reactive or inflammatory conditions.


Patients with biceps tendinitis often complain of a deep, throbbing ache in the anterior shoulder. The pain is usually localized to the bicipital groove and may radiate toward the insertion of the deltoid muscle, or down to the hand in a radial distribution.
This makes it difficult to distinguish from pain that is secondary to impingement or tendinitis of the rotator cuff, or cervical disk disease. Pain from biceps tendinitis usually worsens at night, especially if the patient sleeps on the affected shoulder.
Repetitive overhead arm motion, pulling, or lifting may also initiate or exacerbate the pain.9 The pain is most noticeable in the follow-through of a throwing motion.3 Instability of the tendon may present as a palpable or audible snap when range of motion of the arm is tested.

Rupture of the biceps tendon is one of the most common musculotendinous tears. If the biceps has ruptured, patients will describe an audible, painful popping, followed by relief of symptoms. The anterior shoulder may be bruised, with a bulge visible above the elbow as the muscle retracts distally from the rupture point. Risk factors of biceps rupture include a history of rotator cuff tear, recurrent tendinitis, contralateral biceps tendon rupture, rheumatoid arthritis, age older than 40 years, and poor conditioning.9 If a patient has a feeling of popping, catching, or locking in the shoulder, a SLAP lesion may be present. This usually occurs after trauma, such as a direct blow to the shoulder, a fall on an outstretched arm, or repetitive overhead motion in athletes.

The most common finding of biceps tendon injury is bicipital groove point tenderness.


Many provocative tests (i.e., Yergason, Neer, Hawkins, and Speed tests) have been developed to isolate pathology of the biceps tendonhowever, because these tests create impingement underneath the coracoacromial arch, it is difficult to rule out concomitant rotator cuff lesions.
The Yergason test requires the patient to place the arm at his or her side with the elbow flexed at 90 degrees, and supinate against resistance18 (Figure 2). The test is considered positive if pain is referred to the bicipital groove.

The Neer test involves internal rotation of the arm while in the forward flexed position16 (Figure 3). If the patient experiences pain, it is a positive sign of impingement syndrome.

During the Hawkins test, the patient flexes the elbow to 90 degrees while the physician elevates the patient's shoulder to 90 degrees and places the forearm in a neutral position19 (Figure 4). With the arm supported, the humerus is rotated internally. The test is positive if bicipital groove pain is present.

Speed test, the patient tries to flex the shoulder against resistance with the elbow extended and the forearm supinated9,20 (Figure 5). A positive test is pain radiating to the bicipital groove. If any of these tests is positive, it indicates that impingement is present, which can lead to biceps tendinitis or tendinosis.

Advantages and Disadvantages of Radiologic Imaging Studies in the Evaluation of Biceps Tendinitis

Arthrography (used with MRI or CT to visualize the joint capsule and glenoid labrum)

CT arthrography shows biceps tendon subluxations, ruptures, dislocations, and SLAP lesion
MRI arthrography is preferable for diagnosing biceps lesions and SLAP lesions14 because the agreement between MRI and arthroscopy for biceps lesions is only 37 percent and 60 percent for rotator cuff lesions
Filling of the biceps tendon sheath is unreliable
Sharp images of the tendon may be lost

Ionizing radiation
Bicipital groove view radiography
Shows the width and medial wall angle of the bicipital groove, spurs in the groove, and supertubercular bone spur or ridge
Does not show possible intra-articular disorders of the labrum (soft tissue injuries)

Excellent evaluation of the superior labral complex and biceps tendon
Partial tears of the biceps tendon are more difficult to detect than complete ruptures

Treatment :



Biceps tendinitis or tendinitis may respond to analgesia with nonsteroidal anti-inflammatory drugs (NSAIDs).


Physiotherapy Treatment in Bicipital Tendinitis

Ice, rest from overhead activity, or physical therapy.14 Rehabilitation of an athlete's shoulder involves four phases:

Taping Over Biceps Long And Short Head Also Helpful in Early Stage Of Bicipital Tendinitis.

Taping In Bicipital Tendinitis.

rest; stretching exercises of the scapula, rotator cuff, and posterior capsule;
The goal of stretching is to regain a balanced range of motion without stiffness or pain in any position.

strengthening; and a progressively difficult throwing program.
The patient may begin exercises after the shoulder is pain-free.

Biceps Muscle Strenthening Exercise

Friday, 5 January 2018

Trigeminal Neuralgia and Physiotherapy Treatment :

Trigeminal Nerve

              Trigeminal neuralgia is a chronic pain condition that affects the trigeminal nerve, which carries sensation from your face to your brain. If you have trigeminal neuralgia, even mild stimulation of your face — such as from brushing your teeth or putting on makeup — may trigger a jolt of excruciating pain.

You may initially experience short, mild attacks. But trigeminal neuralgia can progress and cause longer, more-frequent bouts of searing pain. Trigeminal neuralgia affects women more often than men, and it's more likely to occur in people who are older than 50.

Because of the variety of treatment options available, having trigeminal neuralgia doesn't necessarily mean you're doomed to a life of pain. Doctors usually can effectively manage trigeminal neuralgia with medications, injections or surgey


Area Of Trigeminal Nerve
         Illustration showing branches of the trigeminal nerve 
         Trigeminal neuralgia symptoms may include one or more of these patterns:

(1)Episodes of severe, shooting or Tabbing pain that may feel like an electric shock
(2)Spontaneous attacks of pain or attacks triggered by things such as touching the face, chewing, speaking or brushing teeth
Bouts of pain lasting from a few seconds to several minutes
(3)Episodes of several attacks lasting days, weeks, months or longer — some people have periods when they experience no pain
Constant aching, burning feeling that may occur before it evolves into the spasm-like pain of trigeminal neuralgia
(4)Pain in areas supplied by the trigeminal nerve, including the cheek, jaw, teeth, gums, lips, or less often the eye and forehead
Pain affecting one side of the face at a time, though may rarely affect both sides of the face
(5)Pain focused in one spot or spread in a wider pattern


In trigeminal neuralgia, also called tic douloureux, the trigeminal nerve's function is disrupted. Usually, the problem is contact between a normal blood vessel — in this case, an artery or a vein — and the trigeminal nerve at the base of your brain. This contact puts pressure on the nerve and causes it to malfunction.

Trigeminal neuralgia can occur as a result of aging, or it can be related to multiple sclerosis or a similar disorder that damages the myelin sheath protecting certain nerves. Trigeminal neuralgia can also be caused by a tumor compressing the trigeminal nerve.

Some people may experience trigeminal neuralgia due to a brain lesion or other abnormalities. In other cases, surgical injuries, stroke or facial trauma may be responsible for trigeminal neuralgia.


Treatment In Trigeminal Neuralgia


The first line of treatment is medication.
The drug of choice is carbamazepine (Tegretol™), which eliminates or brings acceptable pain relief in 69 percent of patients.
Baclofen (Lioresal™) is the second drug of choice and may be more effective if used with low-dose carbamazepine.
Other medications that may be effective include pimozide, phenytoin (Dilantin™), capsaicin, clonazepam (Klonopin™) and amitriptyline (Elavil™).

Surgical procedures:-

(1)Percutaneous trigeminal radiofrequency rhizotomy
This procedure selectively destroys pain-causing nerve fibers while preserving touch fibers.
Lesioning techniques include radiofrequency thermocoagulation, glycerol injection and mechanical trauma. They are used for patients who are poor candidates for major surgery.
Complications can include weakness in chewing, facial numbness, changes in tearing or salivation and, less often, corneal ulcers, severe aching pain (anesthesia dolorosa) or meningitis.

(2)Microvascular decompression of the trigeminal nerve
This surgical technique involves microsurgery to move the vessel, causing compression away from the trigeminal nerve.
Relief is often long lived; however the incidence of facial numbness is much less than in selective rhizotomy and anesthesia dolorosa does not occur.
The procedure is best for patients younger than 65 with no significant medical or surgical risk factors.
Possible complications include asceptic meningitis, with head and neck stiffness; major neurological problems, including deafness and facial nerve dysfunction; mild sensory loss; cranial nerve palsy, causing double vision, facial weakness, hearing loss; and, on very rare occasions, postoperative bleeding and death.
Microvascular decompression brings complete relief to 75 percent to 80 percent of patients. The recurrence rate is 5 percent to 17 percent.

Physiotherapy Management:-

*The aims of physiotherapy management:- 
To decrease pain and functional limitation, and to improve quality of life.
Treatments include the use of electro-physical agent to relieve pain during acute onset.
Manual therapy, exercise therapy for Temporomandibular Joint (TMJ) as well
as self-massage for facial muscles can also help to restore patients’ functions.
Transcutaneous electrical nerve stimulation (TENS) currently is one of the
most commonly used forms of electroanalgesia

Interferential Therapy (IFT) :

Interferential therapy (IFT) is another electro-physical modality commonly used
for pain management in clinical situations. IFT is the application of alternating
medium frequency current (4,000 Hz) with amplitude modulated at low
frequency (0–250 Hz). Several theoretical physiological mechanisms such as
the gate-control theory, increased circulation, descending pain suppression,
block of nerve conduction, and placebo have been proposed in the literature to
support the analgesic effects of IFT reducing pain for patients with trigeminal neuralgia by having them received
fifteen sessions of IFT with treatment duration of thirty minutes. The intensity of
the impulse varied according to patient’s tolerance. The results suggested that
IFT could be considered as one of the electro-physical modalities in reducing
pain for trigeminal neuralgia.

Monday, 1 January 2018

Wryneck ( Torticollis ) : Physiotherapy Treatment


Congenital Wryneck

Torticolis is a condition (otherwise called 'wryneck') in which the infant's head is tilted. The head frequently pivots towards one shoulder and tilts away to the contrary side. The term 'intrinsic' is additionally at some point utilized while depicting torticollis. This implies it is available at or not long after birth. Infants treated right on time with physiotherapy programs for the most part react well to treatment.

Torticollis is a side effect identified with turning or twisting of the neck. A wide range of causes are conceivable. In babies, torticollis for the most part comes about because of damage amid work and conveyance or the newborn child's position in the womb. Less regularly, it is caused by birth surrenders. In more seasoned youngsters, torticollis may come about because of wounds to the neck muscles, regular contaminations, or different causes.

Agonizing fits of the neck muscles may happen.

Sternocleidomustoid Muscle

Different indications might be available, contingent upon the reason. For instance, there might be a delicate lymph hub (organ) if the reason is disease.


Inherent torticolis.

Obtained torticolis.

Life systems:

The ordinary physiologic scope of pivot of the map book on the hub is 25-53 degrees to either side. The transverse tendon is the essential stabilizer of the atlantoaxial joint and averts unnecessary foremost movement of the chart book on the pivot. It reaches out behind the lairs, between the average segments of the parallel masses of C1. The combined alar tendons go about as optional stabilizers to anticipate front move. The alar tendons reach out from the parallel part of the caves tip to the average part of the occipital condyles, with a lower partition joining to the average part of the sidelong masses of C1.

The sternocleidomastoid muscle has a sternal and clavicular head. The sternal head is coordinated from the manubrium sterni superiorly, along the side and posteriorly and the clavicular from the average third of the clavicle vertically upward. It races to the mastoid procedure. It empowers an ipsilateral horizontal flexion and a contralateral revolution. The muscle expands the upper piece of the cervical spine and flexes the lower part.

Acquired Torticollis


Solid in over 80% of the cases. Sorts strong torticollis

- Fibromatosis colli: torticollis with obvious mass in the SCM;

- Tightness of the SCM without a clear mass;

Postural torticollis with neither mass or snugness.

Birth injury: aspect separation, tears in the sternocleidomastoid muscle

Intrinsic inconsistencies of the craniovertebral intersection: occipitoatlantal combination or Klippel-Feil disorder.

Sternocleidomastoid tumor.

Visual anomalies.

Intrauterine mechanical variables


Sitting or dozing in an irregular position without satisfactory neck bolster.

Poor stance when taking a gander at a PC screen.

Conveying overwhelming lopsided burdens (for instance, a folder case or shopping pack).

Enabling certain muscles of the neck to be presented to chilly (dozing in a draft).


The turning of your neck (torticollis) happens when your muscles supporting the neck on one side are agonizing.

The agony is as a rule on one side of your neck and solidness of the muscles around there turns the neck to the other side. You may think that its exceptionally troublesome when you attempt to rectify your neck, because of agony. Sporadically, the agony is amidst your neck.

The torment may spread to the back of your make a beeline for your shoulder. The muscles of your influenced side might be delicate. Weight on specific zones may trigger a 'fit' of these muscles. Development of your neck is limited, especially on one side.


An exhaustive neurologic examination ought to be performed, and anteroposterior and sidelong radiographs of the cervical spine ought to be gotten. A CT sweep or MRI of the head and neck is important for any patient with relentless neck torment or with neurologic signs and indications.



Agony executioner are regularly useful. for example,

Paracetamol at quality is frequently adequate.

Calming painkillers.

A more grounded torment executioner such codeine.

A muscle relaxant, for example, diazepam.

Different medicines, for example,


A decent stance.

A firm supporting pad.

Warmth pack.


Stretching Exercise


Delicate scope of movement practices for neck.

Extending of sternocleido mastoid muscle.

Strnengthening works out.

Exercises to support dynamic head development.

Visual following.

Horizontal head tilt.

Treatment ball works out.

Side sitting activities.

Hands and knees.

Stooping to standing.

Helped rolling.

Proped sidelying.

Torticollis treatment at home for babies:

Exercise In Torticollis

The best technique for torticollis treatment is to urge your infant to hand his or her head over the two bearings. This will relax tense neck muscles and fix the free ones. Here are a few activities to attempt:

At the point when your child needs to eat, offer the jug or your bosom in a way that urges your infant to get some distance from the favored side. (Utilize your tyke's want to eat to support him or her along!)

When putting your infant down to rest, position him or her to confront the divider. Since babies like to watch out onto the room, your infant will effectively get some distance from the divider and this will extend the fixed muscles of the neck.

Amid play, draw your child's consideration with toys and sounds to make him or her hand over the two bearings.

Related Article :

Erb's Palsy

Monday, 11 December 2017

Erb's Palsy And Physiotherapy :

Erb's palsy And Physiotherapy Treatment : 

Erb's Palsy From Front And Back Side

Erb's palsy also called Erb's Duchenne palsy is a paralysis of the arm(Upper Limb). This injury is caused mainly Due to injury to the upper group of the arm's main nerves, Mainly the injury of the upper trunk C5–C6 nerves root. These Nerve Root form part of the brachial plexus, Forming the ventral rami of spinal nerves C5–C8 and One thoracic nerve T1. These injuries Occurs most commonly, but not exclusively, from shoulder dystocia during a difficult birth. Depending on the nature of the damage, the paralysis can either resolve on its own over a period of months, necessary  Physiotherapy Treatment or Severe Injury May require surgical Intervention.

Nerve Root Explaination From Cervical Area

The paralysis can be partial or complete; the damage to each nerve can range from bruising to Complete Tear. The most commonly involved Nerve root is C5 (aka Erb's point: the union of C5 - C6 roots) as this is mechanically the furthest point from the force of traction, therefore, the first/most affected Nerve Root. Erb–Duchenne palsy presents as a lower motor neuron Injury with sensibility Loss and vegetative phenomena.

Infant Nerve Root And Brachial Plexus

The most commonly involved nerves are the suprascapular nerve, musculocutaneous nerve, and the axillary nerve.

The signs of Erb's Palsy include loss of sensation in the arm and paralysis or wekness of the deltoid, biceps, and brachialis muscles. "The position of the limb, under such conditions, is by : the arm hangs by the adducted and is rotate internally ; the forearm is in pronation and exntension position. Sholder Abduction, elbow Flexion And Supination is lost Mainly. The resulting Condition Look's Like Postion Also Called "waiter's tip Hand ".

Brachial Plexus

If this injury occurs at early age May Leads to affect development (e.g. as a neonate or infant), it often leaves the patient with Delayed growth in the affected arm with everything from the shoulder through to the fingertips smaller than Compare to Normal arm. This also leaves the patient with Delayed muscular, Nervous & circulatory development. The Delayed of muscule development May leads to the arm being much weaker than a Normal one, and less articulation, with many patients unable to lift the arm above shoulder height, as well as leaving many with a Muscle contracture.

Cause :

Cause Of Erb's Palsy

Dystocia ( Difficult ChildBirth-Labor)
Fracture At Clavicle to Neonates.
Any age following trauma to the head and shoulder.

Waiter's Tip Hand Position

Diagnosis :

Examine The Patient's Arm Position Like Adducted From Sholder, Extended From Eblob Joint And Pronated Position With Weakness or Paralysis Of Deltoid, Brachialis,Biceps Most Commonly.
Further Investigation Is By EMG/NCV Reports Or By MRI Accordingly.

Prognosis Of Erb's Palsy

Treatment :

Treatment in Erb's Palsy

Some babies recover on Gradually With Physiotherapy Treatment however, Patient some may require specialist intervention or Surgical Procedure According To Injury.

Neonatal/pediatric neurosurgery is often required for avulsion Injury. Lesions may heal Naturally Over Time and function Gradually return With Help Of Exercise Therapy.

Physiotherapeutic care is required Mainly to restore muscle Function. Although range of motion is recovered in many children under one year in age, individuals who have not yet healed after this point will rarely gain full function in their arm and may develop Deformity.

The three most common treatments for Unrecovered Erb's Palsy are:
1. Nerve transfers (usually from the opposite arm or limb)
2. Sub Scapularis releases and Latissimus Dorsi Tendon Transfers.

Physiotherapy Treatment :

Physiotherapy Treatment In Erb's Palsy

Assessment Of Patient Mainly Muscle Chart Of Whole Upper Limb And Range Of Motion And RD Test.
Accordingly Design Treatment Plan And Monitoring Progress Report With SD Curve At Every 10 Days Helps Recovery Process Going On.

According to Muscle Chart Strenthening Exercise, Electrical Stimulation, Passive Movement Or Active Assited Exercise Are Design.
Home Exercise Are Teached To Patient's Relative And Deformity Correction Position And Splinting Training Are Also Required.
Mainly Aeroplane Splint Commonly Used But It May Be Vary According To Condition.

Splinting in Erb's Palsy

Aeroplane Splint

Friday, 1 December 2017

Parkinson's Disease

Parkinsonism Disease And Physiotherapy Treatment : 

Parkinson's Disease


Parkinsonism is a clinical disorder portrayed by tremor, bradykinesia, inflexibility, and postural shakiness.

Parkinsonism is any condition that causes a mix of the development anomalies found in Parkinson's ailment —, for example, tremor, moderate development, debilitated discourse or muscle firmness — particularly coming about because of the loss of dopamine-containing nerve cells (neurons). 

Posture In Parkinson's Disease

Life systems:-


Parkinsonism illness (PD) is a degenerative, dynamic issue that influences nerve cells in profound parts of the cerebrum called the basal ganglia and the substantia nigra. Nerve cells in the substantia nigra create the neurotransmitter dopamine and are in charge of handing-off messages that arrangement and control body development. For reasons not yet comprehended, the dopamine-delivering nerve cells of the substantia nigra start to cease to exist in a few people. At the point when 80 percent of dopamine is lost, PD side effects, for example, tremor, gradualness of development, solidness, and adjust issues happen.

Body development is controlled by an unpredictable chain of choices including between associated gatherings of nerve cells called ganglia. Data goes to a focal region of the cerebrum called the striatum, which works with the substantia nigra to send motivations forward and backward from the spinal rope to the mind. The basal ganglia and cerebellum are in charge of guaranteeing that development is done in a smooth, liquid way. 

Stages Of Parkinson's Disease



STAGE ONE - A Person as a rule has mellow manifestations, for example, tremors or shaking in a limb.Change, for example, poor stance, loss of balance,and strange outward appearances.

STAGE TWO - Symptoms influence the two appendages and the two sides of the body. The individual has for the most part has issue strolling or adjusting, and the failure to finish physical errands turns out to be more clear.

STAGE THREE - Symptoms can be extreme and incorporate the powerlessness to walk straight or stand. there is a detectable abating of physical developments.

STAGE FOUR - The capacity to walk is regularly restricted

STAGE FIVE - tHE PERSON is regularly unfit to deal with herself and will most likely be unable to stand or walk .she may require consistent one on one nursing care.


Mind damage

Diffuse Lewy body ailment (a kind of dementia)




Numerous framework decay

Dynamic supranuclear paralysis


Wilson ailment

Different reasons for auxiliary parkinsonism include:

Cerebrum harm caused by anesthesia drugs, (for example, amid surgery)

Carbon monoxide harming

Certain medications used to treat mental scatters or queasiness

Mercury harming and other compound poisonings

Overdoses of opiates

MPTP (a contaminant in some road drugs)


Indications of Parkinson's ailment contrast from individual to individual. They additionally change as the illness advances.

Indications ordinarily start showing up between the ages of 50 and 60. They grow gradually and regularly go unnoticed by family, companions, and even the individual who has them.

The most widely recognized one is tremor.

Tremor. A tremor, or shaking, as a rule starts in an appendage, regularly your hand or fingers. You may see a forward and backward rubbing of your thumb and index finger, known as a pill-moving tremor. One normal for Parkinson's infection is a tremor of your hand when it is casual (very still).

Impeded development (bradykinesia). After some time, Parkinson's sickness may lessen your capacity to move and moderate your development, making basic assignments troublesome and tedious. Your means may end up plainly shorter when you walk, or you may think that its hard to escape a seat. Additionally, you may stall as you endeavor to walk, making it hard to move.

Unbending muscles. Muscle firmness may happen in any piece of your body. The hardened muscles can restrain your scope of movement and cause you torment.

Debilitated stance and adjust. Your stance may wind up plainly stooped, or you may have adjust issues because of Parkinson's malady.

Loss of programmed developments. In Parkinson's malady, you may have a diminished capacity to perform oblivious developments, including flickering, grinning or swinging your arms when you walk.

Discourse changes. You may have discourse issues because of Parkinson's illness. You may talk delicately, rapidly, slur or waver before talking. Your discourse might be all the more a monotone as opposed to with the standard articulations.

Composing changes. It might turn out to be difficult to compose, and you're composing may seem little.


Registered tomography (CT) sweeps of individuals with PD typically seem ordinary.

X-ray has turned out to be more precise in conclusion of the ailment after some time, particularly through iron-delicate T2* and SWI groupings at an attractive field quality of no less than 3T, both of which can exhibit nonattendance of the trademark 'swallow tail' imaging design in the dorsolateral substantia nigra.



Carbidopa-levodopa mixture.

Dopamine agonists.

MAO-B inhibitors.

Catechol-O-methyltransferase (COMT) inhibitors.




Gait Training Exercise

Balancing Exercise


Keep up and enhance levels of capacity and autonomy, which will enhance a man's personal satisfaction

Utilize exercise and development techniques to enhance portability

Rectify and enhance anomalous development examples and stance, where conceivable

Boost muscle quality and joint adaptability

Rectify and enhance stance and adjust, and limit dangers of falls

Keep up a decent breathing example and compelling hack

Instruct the individual with Parkinson's and their carer or relatives

Upgrade the impacts of medication treatment.

Various Exercise In Parkinsonism

Physiotherapy Treatment



Visual prompting – a concentration point to venture over and start step; portions of tape on the floor to start or keep strolling through regions that reason moderating or solidifying

Sound-related prompting – tallying 1-2-3 to start strolling; venturing to the beat of a metronome or particular music at a predefined rhythm to proceed with the cadence of a walk

Consideration – Thinking about making a major stride; influencing a more extensive curve to turn

Proprioceptive prompting – shaking from side to side prepared to start a stage; making one stride in reverse as a sign prepared to then walk advances.